"
Team:Hong Kong HKUST/test17
From 2013.igem.org
(Difference between revisions)
| (19 intermediate revisions not shown) | |||
| Line 68: | Line 68: | ||
<script type="text/javascript" src="http://ajax.googleapis.com/ajax/libs/jquery/1.3.2/jquery.min.js"></script> | <script type="text/javascript" src="http://ajax.googleapis.com/ajax/libs/jquery/1.3.2/jquery.min.js"></script> | ||
| - | <script type="text/javascript" src="https://static.igem.org/mediawiki/2013/9/91/ | + | <script type="text/javascript" src="https://static.igem.org/mediawiki/2013/archive/9/91/20130919135656%21Slide_Show.txt"></script> |
<script type="text/javascript"> | <script type="text/javascript"> | ||
| Line 132: | Line 132: | ||
| - | + | #Slideshow | |
| + | { | ||
| + | position:absolute; | ||
| + | left:225px; | ||
| + | top:0px; | ||
| + | } | ||
| Line 226: | Line 231: | ||
<h3><center><div id="kepala" style="height:121px;width:100%;"><img src="https://static.igem.org/mediawiki/igem.org/c/c7/BANNER1_%281%29.png" style="height:121px;width:100%;align:middle;"></div></center></h3> | <h3><center><div id="kepala" style="height:121px;width:100%;"><img src="https://static.igem.org/mediawiki/igem.org/c/c7/BANNER1_%281%29.png" style="height:121px;width:100%;align:middle;"></div></center></h3> | ||
| - | + | ||
| - | + | ||
<div id="Slideshow"> | <div id="Slideshow"> | ||
<ul> | <ul> | ||
<li> | <li> | ||
| - | <img src="https://static.igem.org/mediawiki/2013/d/db/Slideshow3.jpg" width=" | + | <img src="https://static.igem.org/mediawiki/2013/d/db/Slideshow3.jpg" width="550" height="330" /> |
<p>The fundamental cause of obesity is an energy imbalance between calories consumed and expended. To increase expenditure of fatty acids (calories), HKUST 2013 Team aims to introduce glyoxylate shunt as an artificial futile cycle in HepG2 cell.</p> | <p>The fundamental cause of obesity is an energy imbalance between calories consumed and expended. To increase expenditure of fatty acids (calories), HKUST 2013 Team aims to introduce glyoxylate shunt as an artificial futile cycle in HepG2 cell.</p> | ||
</li> | </li> | ||
<li> | <li> | ||
| - | <img src="https://static.igem.org/mediawiki/2013/d/db/Slideshow4.jpg" width=" | + | <img src="https://static.igem.org/mediawiki/2013/d/db/Slideshow4.jpg" width="550" height="330" /> |
<p>Part 1 Sense Fatty Acids: We investigated four different sensing mechanisms using: 1) fatty acid metabolism regulator protein (FadR), 2) peroxisome proliferator-activated receptor-alpha (PPAR-alpha) 3) liver- fatty acid binding protein (FABP1) and 4) binding immunoglobulin protein, HSP5 or glucose regulated protein (GRP78). The four sensing mechanisms were measured and compared. </p> | <p>Part 1 Sense Fatty Acids: We investigated four different sensing mechanisms using: 1) fatty acid metabolism regulator protein (FadR), 2) peroxisome proliferator-activated receptor-alpha (PPAR-alpha) 3) liver- fatty acid binding protein (FABP1) and 4) binding immunoglobulin protein, HSP5 or glucose regulated protein (GRP78). The four sensing mechanisms were measured and compared. </p> | ||
</li> | </li> | ||
Latest revision as of 15:55, 19 September 2013
<!DOCTYPE html PUBLIC "-//W3C//DTD XHTML 1.1//EN"
"http://www.w3.org/TR/xhtml11/DTD/xhtml11.dtd">
-
The fundamental cause of obesity is an energy imbalance between calories consumed and expended. To increase expenditure of fatty acids (calories), HKUST 2013 Team aims to introduce glyoxylate shunt as an artificial futile cycle in HepG2 cell.
-
Part 1 Sense Fatty Acids: We investigated four different sensing mechanisms using: 1) fatty acid metabolism regulator protein (FadR), 2) peroxisome proliferator-activated receptor-alpha (PPAR-alpha) 3) liver- fatty acid binding protein (FABP1) and 4) binding immunoglobulin protein, HSP5 or glucose regulated protein (GRP78). The four sensing mechanisms were measured and compared.
© Copyright HKUST iGEM Team 2013, All Rights Reserved
