Team:SYSU-China/Project/Design

From 2013.igem.org

Revision as of 08:38, 26 September 2013 by Scelta (Talk | contribs)

ipsc

UPDATE 09/18/2013

suicide gene?

Why suicide gene?

References

[1]Jerry M. Adams and Suzanne Cory,The Bcl-2 Protein Family: Arbiters of Cell Survival ,Science 281, 1322 (1998)

[2]Zhen Xie et al.,Multi-Input RNAi-Based Logic Circuit for Identification of Specific Cancer cells, Science 333, 1307 (2011)

[3]Amotz Nechushtan et al, Conformation of the Bax C-terminus regulates subcellular location and cell death, The EMBO Journal Vol.18 No.9 pp.2330–2341, 1999.

[4]Alan G. Porter and Reiner U. JaÈ nicke, Emerging roles of caspase-3 in apoptosis, Cell Death and Differentiation (1999) 6, 99 -104

[5]Dattananda S. Chelur  and  Martin Chalfie,  Targeted cell killing by reconstituted caspases, PNAS, 2007 , vol. 104 no. 7 2283–2288

[6] Stefan J. Riedl, Martin Renatus et al,Structural Basis for the Inhibition of Caspase-3 by XIAP,Cell, Vol. 104, 791–800,2001

[7] Xuanyong Lu, Matthew Lee et al, High level expression of apoptosis inhibitor in hepatoma cell line expressing Hepatitis B virus, Int. J. Med. Sci. 2005 2(1)

[8]Srinivasa M. Srinivasula et al,Generation of Constitutively Active Recombinant Caspases-3 and -6 by Rearrangement of Their Subunits, J. Biol. Chem. 1998, 273:10107-10111.

[9]N Festjens, T Vanden Berghe et al,RIP1, a kinase on the crossroads of a cell's decision to live or die, Cell Death and Differentiation (2007) 14, 400–410

[10]RIP3, an Energy Metabolism Regulator That Switches TNF-Induced Cell Death from Apoptosis to Necrosis

[11]Nils Holler et al, Fas triggers an alternative, caspase-8−independent cell death pathway using the kinase RIP as effector molecule, Nature Immunology 1, 489 - 495 (2000)

[12] Liming Sun et al,Mixed Lineage Kinase Domain-like Protein Mediates Necrosis Signaling Downstream of RIP3 Kinase,Cell,2012, 148, 213–227

[13]Priscilla E. M. Purnick & Ron Weiss ,The second wave of synthetic biology: from modules to systems Nature Reviews Molecular Cell Biology 10, 410-422,2009

[14]Liming Sun et al,Mixed Lineage Kinase Domain-like Protein Mediates Necrosis Signaling Downstream of RIP3 Kinase,Cell,2012, 148, 213–227

[15]Xiaoqing Sun et al ,RIP3, a Novel Apoptosis-inducing Kinase, THE JOURNAL OF BIOLOGICAL CHEMISTRY, Vol. 274, No. 24, Issue of June 11, pp. 16871–16875, 1999

[16]Shi-Mei Zhuang et al, Apoptin, a Protein Derived from Chicken Anemia Virus, Induces p53-independent Apoptosis in Human Osteosarcoma Cells, Cancer Res 1995;55:486-489,

[17]Claude Backendorf et al, Apoptin: Therapeutic Potential of an Early Sensor of Carcinogenic Transformation, Annu. Rev. Pharmacol. Toxicol. 2008. 48:143–69

[18]Xiao Li et al, Antitumor effects of a recombinant fowlpox virus expressing Apoptin in vivo and in vitro, Int. J. Cancer: 119, 2948–2957 (2006)

[19] Astrid A. A. M. Danen-van Oorschot,Importance of Nuclear Localization of Apoptin for Tumor-specific Induction of Apoptosis, THE JOURNAL OF BIOLOGICAL CHEMISTRY, Vol. 278, No. 30, pp. 27729 –27736, 2003

[20]Lars Guelen et al,TAT-apoptin is efficiently delivered and induces apoptosis in cancer cells, Oncogene (2004) 23, 1153–1165

[21]Su-Xia Han et al,Secretory Transactivating Transcription-apoptin fusion protein induces apoptosis in hepatocellular carcinoma HepG2 cells, World J Gastroenterol ,2008, 14(23): 3642-3649

[22]Kyozo KATO et al,Retroviral transfer of herpes simplex thymidine kinase gene into glioma cells causes targeting of gancyclovir cytotoxic effect, Neurol Med Chir (Tokyo). 1994 ;34(6):339-44.

[23]Marc Mesniland Hiroshi Yamasaki,Bystander Effect in Herpes Simplex Virus-Thymidine Kinase/Ganciclovir Cancer Gene Therapy: Role of Gap-junctional Intercellular Communication, Cancer Res ,2000;60:3989-3999.

[24]BENOIˆT SALOMON et al, A Truncated Herpes Simplex Virus Thymidine Kinase Phosphorylates Thymidine and Nucleoside Analogs and Does Not Cause Sterility in Transgenic Mice, Mol. Cell. Biol. 1995, 15(10):5322.

[25] http://www.clontech.com/CN/Products/Cell_Biology_and_Epigenetics/RNA_Interference/shRNA/Tet-Inducible_shRNA?sitex=10022:22372:US

[26]http://www.clontech.com/CN/Products/Cell_Biology_and_Epigenetics/RNA_Interference/MicroRNA/Tet-Inducible_MicroRNA?sitex=10022:22372:US

[27]Lei S. Qi et al, Repurposing CRISPR as an RNA-Guided Platform for Sequence-Specific Control of Gene Expression, Cell 152, 1173–1183, 2013

[28]clontech,BD™ Tet-Off and Tet-On Gene Expression Systems User Manual

[29]http://www.clontech.com/CN/Products/Inducible_Systems/Tetracycline-Inducible_Expression/Tet-On_3G?sitex=10022:22372:US

[30]Jolanta Szulc et al, A versatile tool for conditional gene expression and knockdown, NATURE METHODS, VOL.3 NO.2,109-116

[31]Yi Li et al,Transcription activator-like effector hybrids for conditional control and rewiring of chromosomal transgene expression, SCIENTIFIC REPORTS, 2 : 897

[32]Luke A. Gilbert et al,CRISPR-Mediated Modular RNA-Guided Regulation of Transcription in Eukaryotes, Cell 154, 442–451, July 18, 2013

[33]http://www.clontech.com/CN/Products/Cell_Biology_and_Epigenetics/RNA_Interference/MicroRNA/Tet-Inducible_MicroRNA?sitex=10022:22372:US

[34]http://www.clontech.com/CN/Products/Cell_Biology_and_Epigenetics/RNA_Interference/shRNA/Tet-Inducible_shRNA?sitex=10022:22372:US.

Sun Yat-Sen University, Guangzhou, China

Address: 135# Xingang Rd.(W.), Haizhu Guangzhou, P.R.China